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The pandemic’s true death toll: millions more than official counts - excess deaths thread - incl. excess deaths from all causes

Excess deaths - "Why is the silence deafening, why?"





This phenomena needs to be seriously investigated now. What is going on?

As a personal observation I know of a family of 5 siblings in their 50s. All with co-morbidities but functioning at jobs and stable.

In 2020 one suddenly died of sepsis. No COVID-19 vaccine was available that year and the Delta variant was a very serious version of COVID-19.

In 2022 a 2nd one died - no vaccine for previous 20 months but multiple COVID-19 job exposures ongoing. Died of sudden cervical artery dissection.

In 2023 one sibling was diagnosed with stage 3 breast cancer.

Also in 2023 one was diagnosed with pancreatic cancer and is terminal.

All have a different diagnosis, all most probably with multiple COVID-19 exposures since 2020. It appears to me that it is possible that COVID-19 is doing what pandemics do...greatly affect the weak. Since COVID-19 is a disease of the circulatory system the cumulative damage may manifest in many different ways.

I know many people blame the vaccine for the excess deaths. This seems a popular theory but with almost no one getting the vaccine in 2023 what is the real explanation for the excess deaths in 2023? Is it repeated COVID-19 illnesses? Repeated COVID-19 illnesses plus lingering affects of the vaccines? Added to mental health issues (suicides) and drug addiction (at least in the US)?

The numbers are staggering and we need to know what is going on.
 
I have taken the original COVID-19 vaccine set and all of the boosters. I have suffered no ill effects from the vaccine that I know of. But - I am not a good barometer because I live a limited social life and wear an N95 mask at indoor venues. I am high risk and have not exposed myself to various circulating pathogens for several years.
 
Is COVID-19 is a disease of the circulatory system? I would say it is a disease of the respiratory system, which puts additional strain on the heart. It also puts a great deal of strain on the immune system, which may then not be as effective in controlling other problems. Covid has a worryingly high post acute phase rate of readmission to hospital and deaths. see Long-term outcomes following hospital admission for COVID-19 versus seasonal influenza: a cohort study


Actually. No. It is the damage to the circulatory system that causes the respiratory problems.

"The starting point for problems is injury to the endothelial layer."

Dr. Michelle Bendeck Heart & Stroke researcher​


Importantly, when the endothelium, which is the lining of the blood vessels, is damaged, it has a domino effect on the entire system. That can cause the vessel walls to stop expanding and contracting smoothly, leading to high blood pressure or reducing blood flow to vital organs. “The starting point for problems is injury to the endothelial layer,” says Dr. Bendeck.

https://www.heartandstroke.ca/artic...ar-system#:~:text=It's the foundation of your ,it's%20under%20threat%20from%20COVID&text=Researc h%20on%20COVID%2D19%20has,involve%20heart%20diseas e%20and%20stroke.


and...please see:

COVID-19 – A vascular disease

Hasan K. Siddiqi,[SUP]a,[/SUP][SUP]b[/SUP] Peter Libby,[SUP]a,[/SUP][SUP]⁎[/SUP] and Paul M Ridker[SUP]a,[/SUP][SUP]b[/SUP]
Author information Copyright and License information PMC Disclaimer

See editorial "Editorial commentary: Vascular injury in acute infections and COVID-19: everything old is new again" in Trends Cardiovasc Med, volume 31 on page 6.
Go to:

Abstract


Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) leads to multi-system dysfunction with emerging evidence suggesting that SARS-CoV-2-mediated endothelial injury is an important effector of the virus. Potential therapies that address vascular system dysfunction and its sequelae may have an important role in treating SARS-CoV-2 infection and its long-lasting effects.

​more....

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7556303/

------------------------------------------

I really think people need to think of COVID-19 as a circulatory disease...i.e. the food supply system (oxygen) to the entire body can be compromised which can lead to all sorts of issues, including respiratory.
 
Heart&Stroke is not a journal and the link adds little to the debate. This quote "says Dr. Swartz. The virus can get in there and inflame the blood vessels.” concerns me, as I have seen nothing that leads me to believe that viral replication is taking place in the walls of blood vessels to any great extent. Also I have no idea who Dr. Swartz is, there seem to be lots of Dr. Swartz listed.

The second link is to a peer reviewed journal and explains that it is the primary infection in the respiratory tract which causes the release of cytokines which in turn increase the permeability of the endothelium. This is a common effect and useful, in moderation, as it eases the passage of neutrophils through the barrier.
see
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9221661/
and

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7240244/
 
I am not going to argue about this. The simple truth is that COVID-19 can damage many diverse tissues in the human body. Call it whatever you want.
 
Latest US excess data from the CDC:

https://www.cdc.gov/nchs/nvss/vsrr/COVID19/


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If you look at the column entitled "Percent of Expected Deaths" you can see what percentage of the last week's deaths were expected. i.e. 110% means that the actual deaths were 10% higher than expected.

So for the week of 8/26/2023 there were 12% more deaths as a percentage of the total deaths of 56,393. This is 6,767 more deaths than expected for that week.

-------------------------------------------------------------------------------------------------

Year-to-date chart (this number will change as the last couple of months of data is added)


Click image for larger version  Name:	excessdeathsyearly.png Views:	1 Size:	51.0 KB ID:	983504



Above is the yearly data. So far for 2023 the excess deaths are 1% of total deaths which is an additional 28,691 deaths.
 
Last edited:
Deaths by Week and State


I am displaying the monthy year-to-date data. November now shows as 4% more deaths than expected which is 9,754 more people died than expected. December's full data is still pending.


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Provisional Death Counts for COVID-19
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PAGE 2 of 7
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Table of Contents

Note: Provisional death counts are based on death certificate data received and coded by the National Center for Health Statistics as of December 28, 2023. Death counts are delayed and may differ from other published sources (see Technical Notes). Counts will be updated Thursdays by 5pm.

The provisional counts for COVID-19 deaths are based on a current flow of mortality data in the National Vital Statistics System. National provisional counts include deaths occurring within the 50 states and the District of Columbia that have been received and coded as of the date specified. It is important to note that it can take several weeks for death records to be submitted to National Center for Health Statistics (NCHS), processed, coded, and tabulated. Therefore, the data shown on this page may be incomplete, and will likely not include all deaths that occurred during a given time period, especially for the more recent time periods. Death counts for earlier weeks are continually revised and may increase or decrease as new and updated death certificate data are received from the states by NCHS. COVID-19 death counts shown here may differ from other published sources, as data currently are lagged by an average of 1–2 weeks.

The provisional data presented on this page include the provisional counts of deaths in the United States due to COVID-19, deaths from all causes and percent of expected deaths (i.e., number of deaths received over number of deaths expected based on data from previous years), pneumonia deaths (excluding pneumonia deaths involving influenza), pneumonia deaths involving COVID-19, influenza deaths, and deaths involving pneumonia, influenza, or COVID-19; by week ending date, month, and year, and specific jurisdictions.

For the Index of Provisional COVID-19 Mortality Surveillance and Ad-hoc Data Files, click here. Table 1 has counts of deaths involving COVID-19 and other select causes of death by time-period in which the death occurred. For data on deaths involving COVID-19 by time-period and jurisdiction,
socrata-icon.png
Click here to download.

NOTE: Empty data cells represent death counts between 1-9 that have been suppressed in accordance with NCHS confidentiality standards. Number of deaths reported in this table are the total number of deaths received and coded as of the date of analysis and may not represent all deaths that occurred in that period. Counts of deaths occurring before or after the reporting period are not included in the table. Data during recent periods are incomplete because of the lag in time between when the death occurred and when the death certificate is completed, submitted to NCHS and processed for reporting purposes. This delay can range from 1 week to 8 weeks or more, depending on the jurisdiction and cause of death. The United States population, based on 2021 census estimates from the U.S. Census Bureau, is 331,893,745. United States death counts include the 50 states, plus the District of Columbia and New York City. New York state estimates exclude New York City.

[1] Deaths with confirmed or presumed COVID-19, coded to ICD–10 code U07.1.

[2] Percent of expected deaths is the number of deaths for all causes for these time-periods in 2020-2023 compared to the average number across the same time-period in 2017–2019. Previous analyses of 2015–2016 provisional data completeness have found that completeness is lower in the first few weeks following the date of death (<25%), and then increases over time such that data are generally at least 75% complete within 8 weeks of when the death occurred (8). Recent improvements in the timeliness of provisional mortality data suggest that the data are at least 94% complete within 8 weeks of when the death occurred, see Technical Notes.

[3] Counts of deaths involving pneumonia (J12.0-J18.9) include pneumonia deaths that also involve COVID-19 and exclude pneumonia deaths involving influenza.

[4] Counts of deaths involving influenza (J09-J11) include deaths with pneumonia or COVID-19 also listed as a cause of death.

[5] Deaths with confirmed or presumed COVID-19, pneumonia, or influenza, coded to ICD–10 codes U07.1 or J09–J18.9.
Understanding the Numbers: Provisional Death Counts and COVID-19

Provisional death counts deliver the most complete and accurate picture of lives lost to COVID-19. They are based on death certificates, which are the most reliable source of data and contain information not available anywhere else, including comorbid conditions, race and ethnicity, and place of death.

How it Works


The National Center for Health Statistics (NCHS) uses incoming data from death certificates to produce provisional COVID-19 death counts. These include deaths occurring within the 50 states and the District of Columbia.

NCHS also provides summaries that examine deaths in specific categories and in greater geographic detail, such as deaths by county and by race and Hispanic origin.

COVID-19 deaths are identified using a new ICD–10 code. When COVID-19 is reported as a cause of death – or when it is listed as a “probable” or “presumed” cause — the death is coded as U07.1. This can include cases with or without laboratory confirmation. Why These Numbers are Different


Provisional death counts may not match counts from other sources, such as media reports or numbers from county health departments. Counts by NCHS often track 1–2 weeks behind other data.
  • Death certificates take time to be completed. There are many steps to filling out and submitting a death certificate. Waiting for test results can create additional delays.
  • States report at different rates. Currently, 63% of all U.S. deaths are reported within 10 days of the date of death, but there is significant variation between states.
  • Other reporting systems use different definitions or methods for counting deaths.
Things to know about the data


Provisional counts are not final and are subject to change. Counts from previous weeks are continually revised as more records are received and processed.

Provisional data are not yet complete. Counts will not include all deaths that occurred during a given time period, especially for more recent periods. However, we can estimate how complete our numbers are by looking at the average number of deaths reported in previous years.

Death counts should not be compared across states. Some states report deaths on a daily basis, while other states report deaths weekly or monthly. State vital record reporting may also be affected or delayed by COVID-19 related response activities.

For more detailed technical information, visit the Provisional Death Counts for COVID-19 Technical Notes page.

Download PDF Version


Contents
  1. COVID-19 Mortality Overview
  2. Deaths by Week and State
  3. Deaths by Select Demographic and Geographic Characteristics
  4. Health Disparities
  5. Excess Deaths Associated with COVID-19
  6. Index of Available Data Files
  7. Technical Notes
Last Reviewed: December 28, 2023​

https://www.cdc.gov/nchs/nvss/vsrr/COVID19/
 
Is COVID-19 is a disease of the circulatory system? I would say it is a disease of the respiratory system, which puts additional strain on the heart. It also puts a great deal of strain on the immune system, which may then not be as effective in controlling other problems. Covid has a worryingly high post acute phase rate of readmission to hospital and deaths. see Long-term outcomes following hospital admission for COVID-19 versus seasonal influenza: a cohort study



Viral persistence in children infected with SARS-CoV-2: current evidence and future research strategies

snip

SARS-CoV-2 is known to invade both the respiratory and non-respiratory tissues, causing an infection varying in severity, from asymptomatic or mild, to severe and fatal. Several autopsy studies have documented the anatomopathological findings and related immune changes of multiple organs in patients with critical disease. These findings have shown that COVID-19 is not simply a respiratory infection, but that it potentially has major effects on the whole body, including the endothelial system.

https://www.thelancet.com/journals/lanmic/article/PIIS2666-5247(23)00115-5/fulltext
 
"Although the biological significance of the possibility for viral spread and persistence in children is unknown, the substantial evidence for it should not be neglected, and should inform future clinical, biological, and pharmacological studies."
From the paper's conclusions, note the word possibility.
This paper, like the earlier paper, is working with vRNA detection by PCR. I am aware of a few papers, like this one from the references ( https://www.thelancet.com/journals/lanchi/article/PIIS2352-4642(20)30257-1/fulltext ) see fig. 3, which show virions in tissues away from the respiratory system. These seem to be in fatal cases and more the exception than the rule. Most postmortem's show immune damage but not virions. This was looked at in neurological damage, with the olfactory nerve as a possible conduit, and immune modulated increased blood brain barrier permeability did allow RNA through but no histopathological evidence of viral infection.
In all I remain open to further evidence convincing me of widespread viral infection but, on findings to date, it seems the overwhelming bulk of virally infected cells remain in the respiratory tract and systemic damage is the result of an excessive immune response. APCs can preserve antigen for a long time and carry them all over the body, so inferring viral replication at sites of RNA detection should be done with caution.
 
The Lancet Regional Health - Europe

Volume 36, January 2024, 100802

Comment


Excess mortality in England post COVID-19 pandemic: implications for secondary prevention

Author links open overlay panelJonathan Pearson-Stuttard [SUP]a[/SUP] [SUP]b[/SUP], Sarah Caul [SUP]c[/SUP], Stuart McDonald [SUP]a[/SUP] [SUP]d[/SUP], Emily Whamond [SUP]e[/SUP], John N. Newton [SUP]e[/SUP] [SUP]f[/SUP]
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https://doi.org/10.1016/j.lanepe.2023.100802Get rights and content
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Many countries, including the UK, have continued to experience an apparent excess of deaths long after the peaks associated with the COVID-19 pandemic in 2020 and 2021.[SUP]1[/SUP][SUP],[/SUP][SUP]2[/SUP] Numbers of excess deaths estimated in this period are considerable. The UK Office for National Statistics (ONS) has calculated that there were 7.2% or 44,255 more deaths registered in the UK in 2022 based on comparison with the five-year average (excluding 2020).[SUP]1[/SUP] This persisted into 2023 with 8.6% or 28,024 more deaths registered in the first six months of the year than expected.[SUP]1[/SUP] The Continuous Mortality Investigation (CMI) found a similar excess (28,500 deaths) for the same period using different methods.[SUP]3[/SUP] Several methods can be used to estimate excess deaths, each with limitations which should be considered in interpretation, however the overall trends tend to be consistent across the various methods.

The causes of these excess deaths are likely to be multiple and could include the direct effects of Covid-19 infection,[SUP]1[/SUP] acute pressures on NHS acute services resulting in poorer outcomes from episodes of acute illness,[SUP]4[/SUP] and disruption to chronic disease detection and management.[SUP]5[/SUP] Further analysis by cause and by age- and sex-group may help quantify the relative contributions of these causes.

Since July 2020, the Office for Health Improvement and Disparities (OHID) has published estimates of excess mortality based on a Poisson regression model for England week by week, overall and decomposed by age, ethnicity, region and cause.[SUP]6[/SUP] This model finds that in the period from week ending 3rd June 2022 to 30th June 2023, excess deaths for all causes were relatively greatest for 50–64 year olds (15% higher than expected), compared with 11% higher for 25–49 and < 25 year olds, and about 9% higher for over 65 year old groups. While the median age of these groups has changed since 2020, age-standardised mortality analysis breaking down death rates by sex find clearer age differences still. The age-standardised CMI found similar patterns with the largest relative excess deaths for 2022 observed in young (20–44 years) and middle-aged (45–64 years) adults.[SUP]7[/SUP] These findings should be interpreted carefully because of greater than usual delay in registration of deaths in the latter part of 2022.[SUP]8[/SUP]

Several causes, including cardiovascular diseases, show a relative excess greater than that seen in deaths from all-causes (9%) over the same period (week ending 3rd June 2022–30th June 2023), namely: all cardiovascular diseases (12%), heart failure (20%), ischaemic heart diseases (15%), liver diseases (19%), acute respiratory infections (14%), and diabetes (13%).[SUP]6[/SUP]

For middle-aged adults (50–64) in this 13-month period, the relative excess for almost all causes of death examined was higher than that seen for all ages. Deaths involving cardiovascular diseases were 33% higher than expected, while for specific cardiovascular diseases, deaths involving ischaemic heart diseases were 44% higher, cerebrovascular diseases 40% higher and heart failure 39% higher. Deaths involving acute respiratory infections were 43% higher than expected and for diabetes, deaths were 35% higher. Deaths involving liver diseases were 19% higher than expected for those aged 50–64, the same as for deaths at all ages.[SUP]9[/SUP]

Looking at place of death, from 3rd June 2022 to 30th June 2023 there were 22% more deaths in private homes than expected compared with 10% more in hospitals, but there was no excess in deaths in care homes and 12% fewer deaths than expected in hospices. For deaths involving cardiovascular diseases the relative excess in private homes was higher than all causes at 27%. Deaths in hospital were 8% higher and deaths in care homes only 3% higher.[SUP]9[/SUP]

The greatest numbers of excess deaths in the acute phase of the pandemic were in older adults. The pattern now is one of persisting excess deaths which are most prominent in relative terms in middle-aged and younger adults, with deaths from CVD causes and deaths in private homes being most affected. Timely and granular analyses are needed to describe such trends and so to inform prevention and disease management efforts. Leveraging such granular insights has the potential to mitigate what seems to be a continued and unequal impact on mortality, and likely corresponding impacts on morbidity, across the population.

Contributors


All authors contributed to the design of the paper, JP-S wrote the first draft, SC, SM and JN drafted additional sections, all authors reviewed and commented on drafts.

Declaration of interests


JP-S is Partner at Lane Clark & Peacock LLP, Chair of the Royal Society for Public Health and reports personal fees from Novo Nordisk and Pfizer Ltd outside of this submitted work. SM is Partner at Lane Clark & Peacock LLP and deputy Chair of the Continuous Mortality Investigation. All other authors declare no competing interests.

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https://www.sciencedirect.com/science/article/pii/S2666776223002211?via=ihub
 
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