• FluTrackers.com Inc. does not provide medical advice. Information on this web site is collected from various internet resources, and the FluTrackers board of directors makes no warranty to the safety, efficacy, correctness or completeness of the information posted on this site by any author or poster. The information collated here is for instructional and/or discussion purposes only and is NOT intended to diagnose or treat any disease, illness, or other medical condition. Every individual reader or poster should seek advice from their personal physician/healthcare practitioner before considering or using any interventions that are discussed on this website. By continuing to access this website you agree to consult your personal physican before using any interventions posted on this website, and you agree to hold harmless FluTrackers.com Inc., the board of directors, the members, and all authors and posters for any effects from use of any medication, supplement, vitamin or other substance, device, intervention, etc. mentioned in posts on this website, or other internet venues referenced in posts on this website.
  • We are not asking for any donations. Do not donate to any entity who says they are raising funds for us.

J Immunol Res . Analysis of Lymphocyte Subpopulations and Cytokines in COVID-19-Associated Pneumonia and Community-Acquired Pneumonia

tetano

Editor, Senior Moderator
J Immunol Res


. 2021 Jun 9;2021:6657894.
doi: 10.1155/2021/6657894. eCollection 2021.
Analysis of Lymphocyte Subpopulations and Cytokines in COVID-19-Associated Pneumonia and Community-Acquired Pneumonia


Guohong Liu[SUP] 1 [/SUP], Xianghu Jiang[SUP] 2 [/SUP], Xiaojiao Zeng[SUP] 2 [/SUP], Yunbao Pan[SUP] 2 [/SUP], Haibo Xu[SUP] 1 [/SUP]



Affiliations

Abstract

Background: The 2019 novel coronavirus SARS-CoV-2 caused large outbreaks of COVID-19 worldwide. COVID-19 resembles community-acquired pneumonia (CAP). Our aim was to identify lymphocyte subpopulations to distinguish between COVID-19 and CAP.
Methods: We compared the peripheral blood lymphocytes and their subsets in 296 patients with COVID-19 and 130 patients with CAP. Parameters for independent prediction of COVID-19 were calculated by logistic regression.
Results: The main lymphocyte subpopulations (CD3[SUP]+[/SUP]CD4[SUP]+[/SUP], CD16[SUP]+[/SUP]CD56[SUP]+[/SUP], and CD4[SUP]+[/SUP]/CD8[SUP]+[/SUP] ratio) and cytokines (TNF-α and IFN-γ) of COVID-19 patients were significantly different from that of CAP patients. CD16[SUP]+[/SUP]CD56[SUP]+[/SUP]%, CD4[SUP]+[/SUP]/CD8[SUP]+[/SUP]ratio, CD19[SUP]+[/SUP], and CD3[SUP]+[/SUP]CD4[SUP]+[/SUP] were identified as predictors of COVID-19 diagnosis by logistic regression. In addition, the CD3[SUP]+[/SUP]CD4[SUP]+[/SUP]counts, CD3[SUP]+[/SUP]CD8[SUP]+[/SUP] counts, andTNF-α are independent predictors of disease severity in patients.
Conclusions: Lymphopenia is an important part of SARS-CoV-2 infection, and lymphocyte subsets and cytokines may be useful to predict the severity and clinical outcomes of the disease.
 
Back
Top Bottom