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Emerg Infect Dis. Swine Influenza Virus A (H3N2) Infection in Human, Kansas, USA, 2009

Giuseppe

Emeritus
[Source: Emerg Infect Dis., full text: (LINK). Extract, edited.]


Volume 17, Number 6?June 2011
Letter

Swine Influenza Virus A (H3N2) Infection in Human, Kansas, USA, 2009



Chad M. Cox, Daniel Neises, Rebecca J. Garten, Bill Bryant, Richard A. Hesse, Gary A. Anderson, Ingrid Trevino-Garrison, Bo Shu, Stephen Lindstrom, Alexander I. Klimov, and Lyn Finelli

Author affiliations: Centers for Disease Control and Prevention, Atlanta, Georgia, USA (C.M. Cox, R.J. Garten, B. Shu, S. Lindstrom, A.I. Klimov, L. Finelli); Kansas Department of Health and Environment, Topeka, Kansas, USA (D. Neises, I. Trevino-Garrison); Kansas Animal Health Department, Topeka (B. Bryant); and Kansas State Veterinary Diagnostic Laboratory, Manhattan, Kansas, USA (R.A. Hesse, G.A. Anderson)

Suggested citation for this article


To the Editor:

Triple-reassortant swine influenza viruses (SIVs), which contain genes from human, swine, and avian influenza A viruses, have been enzootic among swine herds in the United States since the late 1990s (1). Although uncommon, occasional transmission of triple-reassortant SIVs from swine to humans has occurred (2?4). Before April 2009, only limited, nonsustained human-to-human transmission of SIVs had been reported (5?7). Although an animal source for pandemic (H1N1) 2009 virus has yet to be identified, the pandemic strain resulted from the reassortment of 2 different lineages of SIV (8).
(?)

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