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pH1N1 Deaths in Texas by County - 2013-2014 Influenza Season
Thank you for gathering these important pieces of information to help the public understand the depth of clinical severity for the variant genetics of Pandemic H1N1 during the past 2 weeks.
We've built a collaborative Excel Spreadsheet with Mr. Oliveros' compilation so that this important summary of original source documents may be maintained by a FluTrackers moderator / coordinator.
Feel free to download and modify as necessary.
Then please share the update on this thread with an upload of your file.
FluTrackers does not seem to allow an Excel attachment, so we have placed the spreadsheet at GeneWurx.com,
If you make an update, please generate a PDF or JPG image and post on this thread. If Mr. Oliveros or Mr. Laidback Al are open to generating time series charts, let's create a new worksheet each week inside this documentary Excel Workbook.
Map - pH1N1 fatalities in Texas by county in the last several weeks.
Data is based on the link in post #4 above. Note: The two unborn fatalities from Travis County are not included on the map, nor is the fatality without an associated county location.
Updated Map Jan 07, 2013 - pH1N1 fatalities in Texas by county in the last several weeks.
Data is based on the link in post above. Note: The two unborn fatalities from Travis County are not included on the map, nor is the fatality without an associated county location.
Re: pH1N1 Deaths in Texas by County - 2013-2014 Influenza Season
pH1N1
Data Sparsity Egg Passage Winter 2013
2014-01-18-21:30
At the WeekEnd, Atlanta
A December Void
During the minimal pH1N1 case count in the Northern Hemisphere's 2012 season and post-season, the US CDC demonstrated the experimental importance of Egg Passage strategies. Variant polymorphisms known to drive deep lung involvement in humans were surfaced repeatedly from quasi-species. That egg-based experimentation was considered valuable and was pursued throughout much of 2013, but evidently not to quantify the actual circulating pH1N1 viral population . . . because the production came to a full halt at the moment that the US flu season's high fatality rates became public due to an unexpected re-emergence of pH1N1.
Prior to the beginning of the present US season, experimental devotion was clear with seventeen sequences published on Egg Passaging having sample dates BETWEEN seasons, a much-appreciated and long overdue analysis bed. That high interest from the depositing organisation, however, waned just as the infective period of actionable usefulness began. After the four mid-November US samples from egg-based passages (concentrated six day period), no further information has been provided.
Silence isNOTGolden
Few explanations satisfy the fact of publication quiescence . . .
Few indeed, unless we consider that the intent of this egg-based benchwork may have been to suggest that the dangerous polymorphisms were "artifacts", a managed message stream flowing from these agencies since 2009 via all outbound portals. Even a passing view of their results shows genetic incongruity to that hypothesis. The public health monopoly's campaign to marginalise the experimental evidence with their tiresome, "artifact of egg passaging" explanation cannot be supported by their genetic outputs.
Flashfires of dominantpH1N1 populations carrying accumulations of those dangerous polymorphisms began as early as the summer of 2013 and then impacted with full force in December 2013. The High-CFRUpsilon subclade's polymorphic set re-emerged. As individual geographies moved into the highest fatality and severity yet seen since modern records have been collected, the public health leaders responded by markedly reducing usable surveillance.
Eight Weeks
During the period from November 19, 2013 to today, no sample has been published with a sequence generated upon an Egg-Based Passage strategy.
During this predictable epidemic with geographically-focal high severity and high fatality, the most relevant information has been withheld for eight weeks.
By those we've trusted to protect our children's lives.
During 4 weeks of fatality intensity, only one sequence has been deposited covering the geography and time period of the high intensity events.
Are we able to measure intent from these actions? Are we satisfied with political utility being the primary measure of our public health organisations?
More importantly, is mission failure driving this silence?
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