• FluTrackers.com Inc. does not provide medical advice. Information on this web site is collected from various internet resources, and the FluTrackers board of directors makes no warranty to the safety, efficacy, correctness or completeness of the information posted on this site by any author or poster. The information collated here is for instructional and/or discussion purposes only and is NOT intended to diagnose or treat any disease, illness, or other medical condition. Every individual reader or poster should seek advice from their personal physician/healthcare practitioner before considering or using any interventions that are discussed on this website. By continuing to access this website you agree to consult your personal physican before using any interventions posted on this website, and you agree to hold harmless FluTrackers.com Inc., the board of directors, the members, and all authors and posters for any effects from use of any medication, supplement, vitamin or other substance, device, intervention, etc. mentioned in posts on this website, or other internet venues referenced in posts on this website.
  • We are not asking for any donations. Do not donate to any entity who says they are raising funds for us.

J Chem Inf Model . Impact of early pandemic stage mutations on molecular dynamics of SARS-CoV-2 M pro

tetano

Editor, Senior Moderator
J Chem Inf Model


. 2020 Aug 27.
doi: 10.1021/acs.jcim.0c00634. Online ahead of print.
Impact of early pandemic stage mutations on molecular dynamics of SARS-CoV-2 M [SUP]pro[/SUP]

Olivier Sheik Amamuddy, Gennady M Verkhivker, ?zlem Tastan Bishop

Abstract

The new coronavirus (SARS-CoV-2) is a global threat to the world health and economy. Its dimeric main protease (M[SUP]pro[/SUP]), which is required for the proteolytic cleavage of viral precursor proteins, is a good candidate for drug development owing to its conservation and the absence of a human homolog. Improving our understanding of M[SUP]pro[/SUP] behaviour can accelerate the discovery of effective therapies to reduce mortality. 100 ns all-atom molecular dynamics simulations of 50 mutant M[SUP]pro[/SUP] dimers obtained from filtered sequences from the GISAID database were analysed using RMSD, RMSF, Rg, the averaged betweenness centrality and geometry calculations. The results showed that SARS-CoV-2 M[SUP]pro[/SUP] essentially behaves in a similar manner to its SAR-CoV homolog. However, we report the following new findings from the variants: (1) residues GLY15, VAL157 and PRO184 have mutated more than once in SARS CoV-2; (2) the D48E variant has lead to a novel "TSEEMLN" loop at the binding pocket; (3) inactive apo Mpro does not show signs of dissociation in 100 ns MD; (4) a non-canonical pose for PHE140 widens substrate binding surface; (5) dual allosteric pockets coinciding with various stabilising and functional components of the substrate binding pocket were found to display correlated compaction dynamics; (6) high betweenness centrality values for residues 17 and 128 in all M[SUP]pro[/SUP] samples suggest their high importance in dimer stability - one such consequence has been observed for the M17I mutation whereby on of the N-fingers was highly unstable. (7) Independent coarse-grained Monte Carlo simulations suggest a relationship between rigidity/mutability and enzymatic function. Our entire approach combining database preparation, variant retrieval, homology modelling, Dynamic Residue Network (DRN), relevant conformation retrieval from 1-D kernel density estimates from reaction coordinates, to other existing approaches of structural analysis and data visualisation within the coronaviral M[SUP]pro[/SUP] is also novel and is applicable to other coronaviral proteins
 
Back
Top Bottom