Sally Furniss
Well-known member
J. Virol. doi:10.1128/JVI.00325-08
High temperature (30?C) blocks aerosol but not contact transmission of influenza virus
<nobr>Anice C. Lowen,</nobr> <nobr>John Steel,</nobr> <nobr>Samira Mubareka,</nobr> and <nobr>Peter Palese<sup>*</sup></nobr> Department of Microbiology, and Department of Medicine, Mount Sinai School of Medicine, New York, NY, USA
[SIZE=-1] <sup>*</sup> To whom correspondence should be addressed. Email: peter.palese@mssm.edu<script type="text/javascript"><!-- var u = "peter.palese", d = "mssm.edu"; document.getElementById("em0").innerHTML = '<a href="mailto:' + u + '@' + d + '">' + u + '@' + d + '<\/a>'//--></script>. [/SIZE]
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<table bgcolor="#e1e1e1" cellpadding="0" cellspacing="0" width="100%"> <tbody><tr><td align="left" bgcolor="#ffffff" valign="middle" width="5%">
</td> <th align="left" valign="middle" width="95%">[SIZE=+2] Abstract[/SIZE]</th></tr></tbody></table>
Influenza causes significant morbidity in tropical regions;<sup> </sup>however, unlike in temperate zones, influenza in the tropics<sup> </sup>is not strongly associated with a given season. We have recently<sup> </sup>shown that influenza virus transmission in the guinea pig model<sup> </sup>is most efficient under cold, dry conditions; conditions which<sup> </sup>are rare in the tropics. Herein, we report the lack of aerosol<sup> </sup>transmission at 30?C, and all humidities tested. Conversely,<sup> </sup>transmission via the contact route was equally efficient at<sup> </sup>30?C and 20?C. Our data imply that contact or short-range<sup> </sup>spread predominates in the tropics and offer an explanation<sup> </sup>for the lack of a well-defined, recurrent influenza season affecting<sup> </sup>tropical and subtropical regions of the world.
http://jvi.asm.org/cgi/content/abstract/JVI.00325-08v1?ct=ct
High temperature (30?C) blocks aerosol but not contact transmission of influenza virus
<nobr>Anice C. Lowen,</nobr> <nobr>John Steel,</nobr> <nobr>Samira Mubareka,</nobr> and <nobr>Peter Palese<sup>*</sup></nobr> Department of Microbiology, and Department of Medicine, Mount Sinai School of Medicine, New York, NY, USA
[SIZE=-1] <sup>*</sup> To whom correspondence should be addressed. Email: peter.palese@mssm.edu<script type="text/javascript"><!-- var u = "peter.palese", d = "mssm.edu"; document.getElementById("em0").innerHTML = '<a href="mailto:' + u + '@' + d + '">' + u + '@' + d + '<\/a>'//--></script>. [/SIZE]
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<table bgcolor="#e1e1e1" cellpadding="0" cellspacing="0" width="100%"> <tbody><tr><td align="left" bgcolor="#ffffff" valign="middle" width="5%">
Influenza causes significant morbidity in tropical regions;<sup> </sup>however, unlike in temperate zones, influenza in the tropics<sup> </sup>is not strongly associated with a given season. We have recently<sup> </sup>shown that influenza virus transmission in the guinea pig model<sup> </sup>is most efficient under cold, dry conditions; conditions which<sup> </sup>are rare in the tropics. Herein, we report the lack of aerosol<sup> </sup>transmission at 30?C, and all humidities tested. Conversely,<sup> </sup>transmission via the contact route was equally efficient at<sup> </sup>30?C and 20?C. Our data imply that contact or short-range<sup> </sup>spread predominates in the tropics and offer an explanation<sup> </sup>for the lack of a well-defined, recurrent influenza season affecting<sup> </sup>tropical and subtropical regions of the world.
http://jvi.asm.org/cgi/content/abstract/JVI.00325-08v1?ct=ct
