• FluTrackers.com Inc. does not provide medical advice. Information on this web site is collected from various internet resources, and the FluTrackers board of directors makes no warranty to the safety, efficacy, correctness or completeness of the information posted on this site by any author or poster. The information collated here is for instructional and/or discussion purposes only and is NOT intended to diagnose or treat any disease, illness, or other medical condition. Every individual reader or poster should seek advice from their personal physician/healthcare practitioner before considering or using any interventions that are discussed on this website. By continuing to access this website you agree to consult your personal physican before using any interventions posted on this website, and you agree to hold harmless FluTrackers.com Inc., the board of directors, the members, and all authors and posters for any effects from use of any medication, supplement, vitamin or other substance, device, intervention, etc. mentioned in posts on this website, or other internet venues referenced in posts on this website.
  • We are not asking for any donations. Do not donate to any entity who says they are raising funds for us.

Front Immunol . Conformational bias in SARS-CoV-2 Spike CD4+ T-cell epitope dominance

tetano

Editor, Senior Moderator
Front Immunol


. 2026 Jul 20:17:1857103.
doi: 10.3389/fimmu.2026.1857103. eCollection 2026.
Conformational bias in SARS-CoV-2 Spike CD4+ T-cell epitope dominance

Samuel J Landry[SUP] 1 [/SUP], N Kalaya Steede[SUP] 1 [/SUP], Yali Tiomkin[SUP] 1 [/SUP], Haley Smith[SUP] 1 [/SUP], Ramgopal R Mettu[SUP] 2 [/SUP], Loren Gragert[SUP] 3 [/SUP], Judith H Aberle[SUP] 4 [/SUP], Kevin J Zwezdaryk[SUP] 5 [/SUP], Crystal Zheng[SUP] 6 [/SUP], Jay K Kolls[SUP] 7 [/SUP], Bronwyn M Gunn[SUP] 8 [/SUP], Amelie E Murrell[SUP] 5 [/SUP], Ivy V Trinh[SUP] 5 [/SUP], John S Schieffelin[SUP] 7 [/SUP], James E Robinson[SUP] #[/SUP][SUP] 7 [/SUP], Elizabeth B Norton[SUP] #[/SUP][SUP] 5 [/SUP]


Affiliations
Abstract

Introduction: Epitope-specific T cells provide significant long-lived protection afforded by adaptive immunity to SARS-CoV-2 spike. CD4+ T-cell epitope peptides that are generated by non-ATP-dependent antigen-processing proteases bind with modest specificity to MHC class II molecules in the endo-lysosome. Studies document the influence of antigen-presenting cell type, manner of endocytosis, and antigen conformation on the strength of CD4+ T-cell response. Nevertheless, few studies report changes in epitope dominance due to circumstances of antigen exposure, which could shape proteolytic antigen processing in the class-II pathway because conformational domains limit proteolysis or MHCII binding.
Methods: Processing of SARS-CoV-2 spike was modeled using limited proteolysis of soluble spike trimer, and the effect of spike conformation on CD4+ T-cell epitope dominance was analyzed using IL-2 Elispots responding to two nine-peptide pools from conformationally stable and unstable regions of spike.
Results: Protease-sensitive sites coincided with domain boundaries and other conformationally unstable regions, confirming that structure limits proteolysis. The ratio of CD4+ T-cell response to stable and unstable peptide pools in two non-hospitalized human subjects cohorts distinguished whether exposure was by infection or vaccination.
Discussion: Circumstances of exposure to spike, e.g., spike mRNA vaccination or SARS-CoV-2 infection, could influence populations of antigen presenting cells and their levels of activation, resulting in different patterns of spike fragmentation, peptide loading, and T-cell response. Circumstances of exposure also affect spike conformational changes that contribute to distinct dominance patterns. Thus, epitope dominance patterns potentially indicate exposure history, immune imprinting, and potentially the protectiveness of the CD4+ T-cell response.

Keywords: antigen processing; fusogenic conformational change; immunological imprinting; innate to adaptive immune communication; original antigenic sin.

 
Back
Top