tetano
Editor, Senior Moderator
The latest issue of ECDC?s monthly series on 'Influenza virus characterisation? covers the time period from September to December 2012. It is a follow-up of the previous month's report.
Since 1 September 2012, A(H1N1)pdm09, A(H3N2) and B/Victoria and B/Yamagata lineage influenza viruses have been detected in relatively low numbers in ECDC-affiliated countries. The report summarises the findings as follows:
Type A and type B viruses are co-circulating in approximately equal proportions.
A(H3N2) and A(H1N1)pdm09 viruses have been detected at comparable levels, with a recent rise in the proportion of A(H1N1)pdm09 viruses.
A(H1N1)pdm09 viruses continued to show genetic drift from the vaccine virus, A/California/07/2009, but the vast majority remained antigenically similar to it.
The vast majority of A(H3N2) viruses have been antigenically similar to A/Victoria/361/2011, the vaccine virus for the 2012?13 influenza season.
B/Victoria lineage viruses fell within, and were antigenically similar to, reference cell-propagated viruses of the B/Brisbane/60/2008 genetic clade.
Recent B/Yamagata-lineage viruses fell into two genetic clades: clade 2 represented by B/Estonia/55669/2012, and clade 3 represented by B/Wisconsin/1/2010 (the vaccine component for the 2012?13 influenza season). Clade 2 viruses have predominated over clade 3.
The next influenza virus characterisation report will be published in March 2013 and will cover data on the 2012-2013 influenza season from September 2012 to February 2013.
For further details, download the complete report 'Influenza virus characterisation - Summary Europe, December 2012'.
http://ecdc.europa.eu/en/press/news...t=32e43ee8-e230-4424-a783-85742124029a&ID=823
Since 1 September 2012, A(H1N1)pdm09, A(H3N2) and B/Victoria and B/Yamagata lineage influenza viruses have been detected in relatively low numbers in ECDC-affiliated countries. The report summarises the findings as follows:
Type A and type B viruses are co-circulating in approximately equal proportions.
A(H3N2) and A(H1N1)pdm09 viruses have been detected at comparable levels, with a recent rise in the proportion of A(H1N1)pdm09 viruses.
A(H1N1)pdm09 viruses continued to show genetic drift from the vaccine virus, A/California/07/2009, but the vast majority remained antigenically similar to it.
The vast majority of A(H3N2) viruses have been antigenically similar to A/Victoria/361/2011, the vaccine virus for the 2012?13 influenza season.
B/Victoria lineage viruses fell within, and were antigenically similar to, reference cell-propagated viruses of the B/Brisbane/60/2008 genetic clade.
Recent B/Yamagata-lineage viruses fell into two genetic clades: clade 2 represented by B/Estonia/55669/2012, and clade 3 represented by B/Wisconsin/1/2010 (the vaccine component for the 2012?13 influenza season). Clade 2 viruses have predominated over clade 3.
The next influenza virus characterisation report will be published in March 2013 and will cover data on the 2012-2013 influenza season from September 2012 to February 2013.
For further details, download the complete report 'Influenza virus characterisation - Summary Europe, December 2012'.
http://ecdc.europa.eu/en/press/news...t=32e43ee8-e230-4424-a783-85742124029a&ID=823